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  • Monotropic Expansion
  • 1. Introduction
    • 1.1 Prevailing Deficit Framework
    • 1.2 Purpose and Goals
    • 1.3 Monotropic Expansion Model
    • 1.4 Addressing Accessibility
    • 1.5 Paper Overview
    • 1.6 Positionality and Rationale
  • 2. Model Mechanism
    • 2.1 Anchoring
    • 2.2 Iterative Context Building
    • 2.3 Cognitive Inertia
    • 2.4 Directionality and Precision
    • 2.5 Scalability and Flexibility
  • 3. Neurological Foundation
    • 3.1 Salience Anchoring and Internal Relevance
    • 3.2 Attentional Modulation and Cognitive Inertia
    • 3.3 Predictive Coding and Inside-Out Construction
    • 3.4 Neurodevelopmental Trajectories and Structural Divergence
    • 3.5 Implications for Structural Modeling and Neuroethical Practice
  • 4. Theoretical Alignment
    • 4.1 Monotropism (Murray, Lesser, Lawson, 2005)
    • 4.2 Executive Dysfunction and Attentional Flexibility
    • 4.3. Weak Central Coherence (Frith, 1989)
    • 4.4. Theory of Mind (ToM) and the Assumption of Deficiency
    • 4.5. Language Processing and Internal Narrative
    • 4.6. Trauma, Inertia, and Pattern Reinforcement
    • 4.7. Double Empathy Problem (Milton, 2012)
    • 4.8. DSM-5 Framing and Pathologized Comparison
  • 5. Implications
    • 5.1. Diagnostic Framing and the Myth of Functioning Labels
    • 5.2. Coexisting Neurodivergent Conditions and Inertial Structures
    • 5.3. Rethinking Support and Accommodation
    • 5.4. Therapy Approaches, Cognitive Models, and Ethical Misalignment
    • 5.5. Self-Perception, Identity, and Communication Disconnects
    • 5.6. Social Systems, Education, and Institutional Friction
  • 6. Reframing Autism
    • 6.1. The Structural Model of Divergence
    • 6.2. Moving Beyond Developmental Language
    • 6.3. Implications for Language, Ethics, and Research
  • 7. Conclusion
  • 8. Update Log
  • Contact & Support
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3. Neurological Foundation

The Monotropic Expansion model describes a structurally distinct form of cognition—one that organizes attention through internally anchored salience and expands outward through deliberate contextual layering. While the model is cognitive in framing, its plausibility is strengthened by growing neuroscientific evidence that supports its core mechanisms. This section connects the model to known patterns in neurological development, attentional processing, and functional connectivity, offering a biological foundation that enhances both its coherence and its potential for empirical validation.

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Last updated 2 months ago