shapeofsoup
  • Monotropic Expansion
  • 1. Introduction
    • 1.1 Prevailing Deficit Framework
    • 1.2 Purpose and Goals
    • 1.3 Monotropic Expansion Model
    • 1.4 Addressing Accessibility
    • 1.5 Paper Overview
    • 1.6 Positionality and Rationale
  • 2. Model Mechanism
    • 2.1 Anchoring
    • 2.2 Iterative Context Building
    • 2.3 Cognitive Inertia
    • 2.4 Directionality and Precision
    • 2.5 Scalability and Flexibility
  • 3. Neurological Foundation
    • 3.1 Salience Anchoring and Internal Relevance
    • 3.2 Attentional Modulation and Cognitive Inertia
    • 3.3 Predictive Coding and Inside-Out Construction
    • 3.4 Neurodevelopmental Trajectories and Structural Divergence
    • 3.5 Implications for Structural Modeling and Neuroethical Practice
  • 4. Theoretical Alignment
    • 4.1 Monotropism (Murray, Lesser, Lawson, 2005)
    • 4.2 Executive Dysfunction and Attentional Flexibility
    • 4.3. Weak Central Coherence (Frith, 1989)
    • 4.4. Theory of Mind (ToM) and the Assumption of Deficiency
    • 4.5. Language Processing and Internal Narrative
    • 4.6. Trauma, Inertia, and Pattern Reinforcement
    • 4.7. Double Empathy Problem (Milton, 2012)
    • 4.8. DSM-5 Framing and Pathologized Comparison
  • 5. Implications
    • 5.1. Diagnostic Framing and the Myth of Functioning Labels
    • 5.2. Coexisting Neurodivergent Conditions and Inertial Structures
    • 5.3. Rethinking Support and Accommodation
    • 5.4. Therapy Approaches, Cognitive Models, and Ethical Misalignment
    • 5.5. Self-Perception, Identity, and Communication Disconnects
    • 5.6. Social Systems, Education, and Institutional Friction
  • 6. Reframing Autism
    • 6.1. The Structural Model of Divergence
    • 6.2. Moving Beyond Developmental Language
    • 6.3. Implications for Language, Ethics, and Research
  • 7. Conclusion
  • 8. Update Log
  • Contact & Support
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3.4 Neurodevelopmental Trajectories and Structural Divergence

Neuroanatomical studies of autistic development point to early patterns of accelerated brain growth, followed by atypical synaptic pruning and functional reorganization. Rather than following a trajectory of broad generalization, autistic brains may develop narrow but deep pathways that support recursive reinforcement of meaningful detail.

Courchesne et al. (2011) used MRI studies to demonstrate early brain overgrowth in autistic children, followed by a divergence from typical pruning patterns. This atypical neurodevelopmental sequence mirrors the architecture of Monotropic Expansion. Anchored attention and contextual depth likely reflect not a delay, but a structural divergence—a growth pattern optimized for precision over flexibility, coherence over generalization.

In this light, autistic cognition is not underdeveloped. It is differently sequenced, internally calibrated, and neurologically valid.

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Last updated 2 months ago