shapeofsoup
  • Monotropic Expansion
  • 1. Introduction
    • 1.1 Prevailing Deficit Framework
    • 1.2 Purpose and Goals
    • 1.3 Monotropic Expansion Model
    • 1.4 Addressing Accessibility
    • 1.5 Paper Overview
    • 1.6 Positionality and Rationale
  • 2. Model Mechanism
    • 2.1 Anchoring
    • 2.2 Iterative Context Building
    • 2.3 Cognitive Inertia
    • 2.4 Directionality and Precision
    • 2.5 Scalability and Flexibility
  • 3. Neurological Foundation
    • 3.1 Salience Anchoring and Internal Relevance
    • 3.2 Attentional Modulation and Cognitive Inertia
    • 3.3 Predictive Coding and Inside-Out Construction
    • 3.4 Neurodevelopmental Trajectories and Structural Divergence
    • 3.5 Implications for Structural Modeling and Neuroethical Practice
  • 4. Theoretical Alignment
    • 4.1 Monotropism (Murray, Lesser, Lawson, 2005)
    • 4.2 Executive Dysfunction and Attentional Flexibility
    • 4.3. Weak Central Coherence (Frith, 1989)
    • 4.4. Theory of Mind (ToM) and the Assumption of Deficiency
    • 4.5. Language Processing and Internal Narrative
    • 4.6. Trauma, Inertia, and Pattern Reinforcement
    • 4.7. Double Empathy Problem (Milton, 2012)
    • 4.8. DSM-5 Framing and Pathologized Comparison
  • 5. Implications
    • 5.1. Diagnostic Framing and the Myth of Functioning Labels
    • 5.2. Coexisting Neurodivergent Conditions and Inertial Structures
    • 5.3. Rethinking Support and Accommodation
    • 5.4. Therapy Approaches, Cognitive Models, and Ethical Misalignment
    • 5.5. Self-Perception, Identity, and Communication Disconnects
    • 5.6. Social Systems, Education, and Institutional Friction
  • 6. Reframing Autism
    • 6.1. The Structural Model of Divergence
    • 6.2. Moving Beyond Developmental Language
    • 6.3. Implications for Language, Ethics, and Research
  • 7. Conclusion
  • 8. Update Log
  • Contact & Support
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3.2 Attentional Modulation and Cognitive Inertia

Another key element of the model is cognitive inertia: the mental effort required to redirect focus once a contextual map is underway. This inertia can be understood as a structural feature of attentional networks. The frontoparietal control network, responsible for task switching and adaptive focus, often exhibits atypical connectivity in autistic individuals—particularly in its coordination with the salience network and sensory processing regions.

Research by Uddin et al. (2016) demonstrates reduced functional connectivity between the frontoparietal network and the salience network in autistic individuals, directly contributing to the increased cognitive cost of attentional shifting. When a mental model is already expanding from a fixed anchor, interruption requires a full reset: disengaging from the current trajectory, discarding partially formed context, and establishing a new internal reference point. Rather than indicating executive dysfunction, this inertia reflects a structural inefficiency tied to coherence preservation—not control failure.

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