shapeofsoup
  • Monotropic Expansion
  • 1. Introduction
    • 1.1 Prevailing Deficit Framework
    • 1.2 Purpose and Goals
    • 1.3 Monotropic Expansion Model
    • 1.4 Addressing Accessibility
    • 1.5 Paper Overview
    • 1.6 Positionality and Rationale
  • 2. Model Mechanism
    • 2.1 Anchoring
    • 2.2 Iterative Context Building
    • 2.3 Cognitive Inertia
    • 2.4 Directionality and Precision
    • 2.5 Scalability and Flexibility
  • 3. Neurological Foundation
    • 3.1 Salience Anchoring and Internal Relevance
    • 3.2 Attentional Modulation and Cognitive Inertia
    • 3.3 Predictive Coding and Inside-Out Construction
    • 3.4 Neurodevelopmental Trajectories and Structural Divergence
    • 3.5 Implications for Structural Modeling and Neuroethical Practice
  • 4. Theoretical Alignment
    • 4.1 Monotropism (Murray, Lesser, Lawson, 2005)
    • 4.2 Executive Dysfunction and Attentional Flexibility
    • 4.3. Weak Central Coherence (Frith, 1989)
    • 4.4. Theory of Mind (ToM) and the Assumption of Deficiency
    • 4.5. Language Processing and Internal Narrative
    • 4.6. Trauma, Inertia, and Pattern Reinforcement
    • 4.7. Double Empathy Problem (Milton, 2012)
    • 4.8. DSM-5 Framing and Pathologized Comparison
  • 5. Implications
    • 5.1. Diagnostic Framing and the Myth of Functioning Labels
    • 5.2. Coexisting Neurodivergent Conditions and Inertial Structures
    • 5.3. Rethinking Support and Accommodation
    • 5.4. Therapy Approaches, Cognitive Models, and Ethical Misalignment
    • 5.5. Self-Perception, Identity, and Communication Disconnects
    • 5.6. Social Systems, Education, and Institutional Friction
  • 6. Reframing Autism
    • 6.1. The Structural Model of Divergence
    • 6.2. Moving Beyond Developmental Language
    • 6.3. Implications for Language, Ethics, and Research
  • 7. Conclusion
  • 8. Update Log
  • Contact & Support
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6.2. Moving Beyond Developmental Language

Much of the current language surrounding autism frames it as a developmental disorder, implying that it reflects a deviation from an expected progression toward a neurotypical norm. This framing is both conceptually flawed and functionally misleading.

What develops is not autism itself, but the external presentation of autism in a world that either misaligns with or accommodates monotropic cognition. Autism, understood through the Monotropic Expansion model, is already present in structure from the outset. The intensity, adaptability, or visibility of traits may shift over time, but the directional foundation of cognition remains intact.

Describing autism as “developmental” conflates emergent behavioral complexity with intrinsic structural difference. It suggests that autism is something that must be corrected or caught up, rather than understood on its own terms. This perspective fuels misdiagnosis, failed interventions, and stigma—not because it names autism, but because it names it wrong.

To move forward, both research and public discourse must reject pathologizing timelines in favor of structural clarity. Autism is not “late.” It is structurally different. It does not fall behind—it expands outward from an anchor that was never in the same place to begin with.

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Last updated 2 months ago